Abstract Afterload enhancement (AE) of rat engineered heart tissue (EHT) in vitro leads to a multitude of changes that in vivo are referred to as pathological cardiac hypertrophy: e. g. cardiomyocyte hypertrophy. contractile dysfunction. https://www.chiggate.com/l-oreal-preference-permanent-hair-colour-4-tahiti-dark-brown-sale/
Blockade of miR-140-3p prevents functional deterioration in afterload-enhanced engineered heart tissue
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